浴室傳播軍團(tuán)菌血清檢測試劑盒

廣州健侖生物科技有限公司

廣州健侖長期供應(yīng)：軍團(tuán)菌、諾如病毒、流感病毒等傳染病系列的快速檢測試劑盒。

軍團(tuán)菌的檢測試劑盒包括：軍團(tuán)菌尿液抗原檢測試劑盒、軍團(tuán)菌抗體快速檢測卡（膠體金法）、軍團(tuán)菌抗原快速檢測卡（膠體金法）、軍團(tuán)菌水樣檢測試劑盒、軍團(tuán)菌乳膠凝集試劑盒（軍團(tuán)菌診斷血清）、嗜肺軍團(tuán)菌核酸熒光PCR檢測試劑盒。

我司還提供其它進(jìn)口或國產(chǎn)試劑盒：包括傳染病系列、免疫組化系列、診斷血清等產(chǎn)品。

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浴室傳播軍團(tuán)菌血清檢測試劑盒

實驗步驟

1） 將所有的材料和樣品都平衡至室溫（2-30℃）

2） 將所有的檢測卡從密封的試劑袋中取出。

3） 將樣品點滴器垂直置于樣品孔上方，向樣品孔中加入3滴樣品（120-150ul）。

4） 10分鐘內(nèi)讀取結(jié)果，強陽性樣品可能會早點出現(xiàn)結(jié)果。

注意：10分鐘后讀取的實驗結(jié)果可能會不準(zhǔn)確。

結(jié)果說明

陽性結(jié)果：檢測線區(qū)域出現(xiàn)明顯的粉色條帶，另外質(zhì)控線區(qū)域出現(xiàn)粉色條帶。

陰性結(jié)果：檢測線區(qū)域不顯色，質(zhì)控線區(qū)域出現(xiàn)明顯的粉色條帶。

無效結(jié)果：靠近檢測線的質(zhì)控線在加樣品后15分鐘內(nèi)不可見的話，則實驗結(jié)果無效。

7、產(chǎn)品特點
★操作簡便，無需其它儀器和試劑，易于在各級醫(yī)院推廣；
★反應(yīng)迅速，5分鐘內(nèi)即可得到結(jié)果；
★結(jié)果清晰，易于判定；
★敏感度高，特異性強。

想了解更多的產(chǎn)品及服務(wù)請掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司

【市 場 部】    楊永漢

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【騰訊Q Q】 2042552662

【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-103室

眶上裂與眶尖綜合征，顱內(nèi)動脈瘤，顱內(nèi)腫瘤，其它如結(jié)核、霉菌、梅毒與化膿性炎癥引起的顱底腦膜炎等。由于病細(xì)菌不同，其發(fā)病機理亦各不相同，如腫瘤的直接壓迫所致，原發(fā)性炎癥時，動眼、滑車與外展神經(jīng)纖維呈脫髓鞘改變等。1、動眼神經(jīng)麻痹表現(xiàn)為上瞼下垂，眼球外斜，向上外、上內(nèi)、下內(nèi)、 同側(cè)方向運動障礙，瞳孔散大，對光反應(yīng)及調(diào)節(jié)反應(yīng)消失，頭向健側(cè) 歪斜。*性癱瘓多為周圍性，而不*性多為核性。2、滑車神經(jīng)麻痹表現(xiàn)為眼球不能向下外方向運動，伴有復(fù)視，下樓時 復(fù)視明顯，致使下樓動作十分困難。頭呈特殊位，呈下頦向下頭面向 健側(cè)的姿勢。3、外展神經(jīng)麻痹表現(xiàn)為眼內(nèi)斜視，不能外展，并有復(fù)視。（一）核性 及束性麻痹 細(xì)菌動眼神經(jīng)核在中腦占據(jù)的范圍較大，故核性損害多 引起不全麻痹，且多為兩側(cè)性，可見有神經(jīng)梅毒，臘腸中毒及白喉等 。束性損害多引起一側(cè)動眼神經(jīng)麻痹，表現(xiàn)為同側(cè)瞳孔擴大，調(diào)節(jié)機 能喪失及瞼下垂，眼球被外直肌及上斜肌拉向外側(cè)并稍向下方。1、腦干腫瘤:特征的臨床表現(xiàn)為出現(xiàn)交叉性麻痹，即病變節(jié)段同側(cè)的 核及核下性顱神經(jīng)損害及節(jié)段下對側(cè)的錐體束征。顱神經(jīng)癥狀細(xì)菌病 變節(jié)段水平和范圍不同而異。如中腦病變多表現(xiàn)為病變側(cè)動眼神經(jīng)麻 痹，橋腦病變可表現(xiàn)為病變側(cè)眼球外展及面神經(jīng)麻痹，同側(cè)面部感覺 障礙以及聽覺障礙。延髓病變可出現(xiàn)病變側(cè)舌肌麻痹、咽喉麻痹、舌 后1/3味覺消失等。腦干誘發(fā)電位、CT、MRI可明確診斷。2、腦干損傷:多有明確的外傷史,傷后長時間的昏迷,且有眼球運動障 礙等,診斷不難。3、顱底骨折:顱腦外傷后可損傷頸內(nèi)動脈，產(chǎn)生頸內(nèi)動脈—海綿竇瘺 出現(xiàn)眼球運動受限和視力減退，同時可有頭部或眶部連續(xù)性雜音，搏 動性眼球突出。1、顱底動脈瘤:動眼神經(jīng)麻痹單獨出現(xiàn)時，常見于顱 底動脈瘤而罕見于其他腫瘤。本病多見于青壯年，多有慢性頭痛及蛛 網(wǎng)膜下腔出血病史，亦可以單獨的動眼神經(jīng)麻痹出現(xiàn)。腦血管造影多 能明確診斷。
Orbital fissure and orbital apex syndrome, intracranial aneurysms, intracranial tumors, other such as tuberculosis, mold, syphilis and purulent inflammation caused by skull base meningitis. Due to the different bacteria, the pathogenesis is also different, such as direct compression of the tumor caused by primary inflammation, oculomotor, pulley and outreach nerve fibers were demyelinated changes. 1, oculomotor nerve paralysis manifested ptosis, eyeball oblique, upward, medial, inferior, ipsilateral dyskinesia, mydriasis, light response and regulatory response disappeared, head to the contralateral skew. Complete paralysis is mostly peripheral, but not complete and mostly nuclear. 2, the performance of the pulley nerve paralysis can not be the downward direction of the eye movement, with diplopia, apparent dip under the stairs, resulting in very difficult action down the stairs. Head was a special bit, was chin downward head facing the contralateral posture. 3, outreach nerve paralysis manifested as intraocular strabismus, can not be outreach, and diplopia. (A) of the nucleus and bundle paralysis bacteria oculomotor nerve nucleus in the brain to occupy a larger range, so more than the nuclear damage caused by incomplete paralysis, and mostly bilateral, showing neurosyphilis, dachshund poisoning and diphtheria. Beam damage caused by more oculomotor nerve paralysis on one side, manifested as ipsilateral pupil dilation, loss of regulatory function and ptosis, the eye was lateral and lateral oblique pull the lateral muscle and slightly downward. 1, brain stem tumors: the clinical manifestations of the characteristics of the emergence of cross-paralysis, ipsilateral lesion of the nucleus and subnuclear cranial nerve damage and contralateral cone pyramidal signs. Cranial neurological symptoms vary in the level and extent of bacterial disease. Such as midbrain lesions showed lesions lateral oculomotor nerve palsy, pons can be manifested as lesion side of the eyeball outreach and facial paralysis, ipsilateral facial sensory disturbances and hearing impairment. Bulbar lesion lesions of the lateral tongue can be paralysis, pharyngolaryngeal paralysis, the tongue after the disappearance of 1/3 taste. Brainstem evoked potential, CT, MRI can confirm the diagnosis. 2, brain stem injury: a clear history of trauma, after a long period of coma, and eye movement disorders, the diagnosis is not difficult. 3, skull fracture: after traumatic brain injury can damage the internal carotid artery, resulting in internal carotid artery - cavernous fistula appear limited eye movement and vision loss, at the same time there may be continuous head and orbital murmur, pulsatile eyeball prominent. 1, skull base aneurysm: oculomotor nerve paralysis alone, common in the skull base aneurysms and rare in other tumors. The disease more common in young adults, and more chronic headache and history of subarachnoid hemorrhage, can also be a separate oculomotor nerve paralysis. Cerebral angiography can definiy diagnose.